A healthy result should fall into the range 0 - 4.1 mcg/mg creatinine.
D-Lactate is produced by bacteria residing in the colon when carbohydrates are not completely absorbed in the small intestine. This by-product is excreted in the urine.
When large amounts are absorbed it can cause:
Although a temporal relationship has been described between elevations of plasma and urine D-lactate and the accompanying encephalopathy (brain disease, damage, or malfunction), the mechanism of neurologic manifestations has not been explained.
D-lactic acidosis is typically observed in patients with short-bowel syndrome and following jejunoileal bypass resulting in carbohydrate malabsorption.
Also, healthy children presenting with gastroenteritis may also develop the critical presentation of D-lactic acidosis.
Routine lactic acid determinations in blood will not reveal abnormalities because most lactic acid assays measure only L-lactate. Accordingly, D-lactate analysis must be specifically requested (eg, DLAC / D-Lactate, Plasma). However, as D-lactate is readily excreted in urine, this is the preferred specimen for D-lactate determinations.
If you have any tendency for carbohydrate malabsorption, even favorable organisms (e.g., L. acidophilus) can grow and lead to increased highly acidic conditions that favor formation of D-Lactate. This condition is revealed by high D-Lactate in urine.
Some possible treatment options (always consult your doctor first before starting any treatment):
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