Phenylalanine (Plasma)

Optimal Result: 31.7 - 71 nmol/ML.

- Low protein or calorie intake, or inflammation can lead to lower levels.

- Nutrient cofactors of phenylalanine catabolism include tetrahydrobiopterin (BH4), nonheme iron, vitamins B6 & B3, copper, niacin, vitamin C, magnesium, SAMe.

- Lower plasma phenylalanine levels seen in depression and associated with lower neurotransmitter levels.

- A higher protein intake or supplementation results in higher levels.

- Elevated levels of phenylalanine compete with other large neutral amino acids.

- Higher phenylalanine levels have been found in type 2 diabetes, and with increased BMI, schizophrenia, and rheumatoid arthritis.

- Inflammation can impair the function of phenylalanine hydroxylase, leading to elevated phenylalanine levels and reduced tyrosine levels. In animal studies cortisol increased the activity of phenylalanine hydroxylase.

- Elevated phenylalanine in PKU has been shown to inhibit HMG-CoA reductase and can result in lower CoQ10 and cholesterol.

What does it mean if your Phenylalanine (Plasma) result is too low?

- Low protein or calorie intake, or inflammation can lead to lower levels.

- Nutrient cofactors of phenylalanine catabolism include tetrahydrobiopterin (BH4), nonheme iron, vitamins B6 & B3, copper, niacin, vitamin C, magnesium, SAMe.

- Lower plasma phenylalanine levels seen in depression and associated with lower neurotransmitter levels.

What does it mean if your Phenylalanine (Plasma) result is too high?

- A higher protein intake or supplementation results in higher levels.

- Elevated levels of phenylalanine compete with other large neutral amino acids.

- Higher phenylalanine levels have been found in type 2 diabetes, and with increased BMI, schizophrenia, and rheumatoid arthritis.

- Inflammation can impair the function of phenylalanine hydroxylase, leading to elevated phenylalanine levels and reduced tyrosine levels. In animal studies cortisol increased the activity of phenylalanine hydroxylase.

- Elevated phenylalanine in PKU has been shown to inhibit HMG-CoA reductase and can result in lower CoQ10 and cholesterol.

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