Cadmium

Optimal Result: 0 - 0.5 mg/kg Dry Wt.

Fecal cadmium (Cd) provides an estimate of very recent oral exposure to the element, and to a much lesser extent the biliary excretion of Cd from the body. Cd absorption is suppressed to some extent with appropriate intake of zinc, calcium, and selenium. Cd is found in varying amounts in foods, from very low for some fruits to high in some shellfish (oysters, anchovies) and organ meats. Root vegetables tend to have higher Cd content than other vegetables. Refined carbohydrates have very little zinc in relation to the Cd. Other sources of Cd include human biosolids, pigments and paints, batteries (Ni-Cd), plastics and synthetic rubber (tires).

Extensive bodily retention of Cd may be symptomatically associated with fatigue, weight loss, osteomalacia, and lumbar pain (Atai-Atai disease). Extensive exposure to Cd may adversely affect the kidneys, lungs, testes, arterial walls, and bones. Chronic Cd excess may lead to microcytic, hypochromic anemia, proteinuria, and functional zinc deficiency.

Urinary or serum beta-2-microglobulin is considered to be the best test for assessment of Cd-associated functional damage to the kidneys.

What does it mean if your Cadmium result is too high?

Fecal cadmium (Cd) provides an estimate of very recent oral exposure to the element, and to a much lesser extent the biliary excretion of Cd from the body. Cd absorption is suppressed to some extent with appropriate intake of zinc, calcium, and selenium. Cd is found in varying amounts in foods, from very low for some fruits to high in some shellfish (oysters, anchovies) and organ meats. Root vegetables tend to have higher Cd content than other vegetables. Refined carbohydrates have very little zinc in relation to the Cd. Other sources of Cd include human biosolids, pigments and paints, batteries (Ni-Cd), plastics and synthetic rubber (tires).

Extensive bodily retention of Cd may be symptomatically associated with fatigue, weight loss, osteomalacia, and lumbar pain (Atai-Atai disease). Extensive exposure to Cd may adversely affect the kidneys, lungs, testes, arterial walls, and bones. Chronic Cd excess may lead to microcytic, hypochromic anemia, proteinuria, and functional zinc deficiency.

Urinary or serum beta-2-microglobulin is considered to be the best test for assessment of Cd-associated functional damage to the kidneys.

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