Optimal Result:
0 - 3 ug/dL.
Lead toxicity causes paralysis and pain in the extremities due to effects on demyelinization, axonal degeneration, and presynaptic block. Lead toxicity commonly affects sensory, visual, auditory, and cerebellar (coordination) functions, reflecting its impact on the nervous system. Normocytic, sideroblastic anemia is the consequence of lead’s inhibiting effects on enzymes in the heme biosynthesis pathway. Other clinical signs associated with lead toxicity are kidney damage, epigastric pain and nausea, and male and female reproductive failure. Hyperactivity, anorexia, decreased play activity, low intelligence quotient and poor school performance have been observed in children with high lead levels. Sources of lead include lead pipes, painted toys, some red lipsticks, lead paint or its dust, soil around old cars, old homes, or highways (past leaded gasoline contamination).
Whole blood is concentrated about 75-fold greater than that of serum or plasma and it has the highest correlation with toxicity. For this reason, whole blood lead is defined by the CDC as the preferred test for detecting lead exposure. The World Health Organization has defined high whole-blood lead levels as > 20 μg/dL in adults and > 10 μg/dL in children. As of 2007, the CDC acceptable level of whole blood lead was 10 μg/dL. Some have proposed to lower reference values for whole blood lead.
Associated Symptoms and Diseases:
Microcytic hypochromic anemia, renal dysfunction, hypertension, anorexia, muscle discomfort, constipation, metallic taste, low IQ (children)
Sources:
Certain supplements, paint, contaminated soils (and plants grown in them), plumbing
Protective Measures:
Alpha lipoic acidprotection against toxicity, iron (reduces intestinal absorption
Chelating Agents:
Ca –EDTA
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