C. albicans is a human commensal yeast. By penetrating the intestinal barrier this pathogen is able to thrive in the human host. Its inflammatory effect in the gastrointestinal tract opens the intestinal barrier, putting tissue and organs at risk for autoimmunity. Candida has been shown to crossreact with a variety of human tissues and thus, when Candida or its antigens reach the blood stream, the antibodies produced against it may turn on self-tissue proteins. The end result can be autoimmunity.
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Increased risk of gastrointestinal disorders and extra-intestinal autoimmunities.
C. albicans is a human commensal yeast. By penetrating the intestinal barrier this pathogen is able to thrive in the human host. Its inflammatory effect in the gastrointestinal tract opens the intestinal barrier, putting tissue and organs at risk for autoimmunity. Candida has been shown to crossreact with a variety of human tissues and thus, when Candida or its antigens reach the blood stream, the antibodies produced against it may turn on self-tissue proteins. The end result can be autoimmunity.
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Acinetobacter, Aspergillus, Babesia + Ehrlichia + Bartonella, Blastocystis hominis, Borrelia burgdorferi, Campylobacter jejuni, Candida albicans, Chlamydias, Citrullinated EBV, Clostridium difficile, Cryptosporidium, CYP450, mimic Hepatitis C Peptide, Cytomegalovirus, Entamoeba histolytica, Giardia lamblia, Helicobacter pylori, Human + Chlamydia HSP-60, Human Herpesvirus-6, Klebsiella, Mycobacterium avium, Mycoplasmas, Penicillium, Porphyromonas gingivalis, Rotavirus, Stachybotrys chartarum, Streptococcal M Protein, Streptococcus mutans, Streptozymes, Yersinia enterocolitica
